Thursday, November 28, 2019
The Patriot Movie Plot Summary Essay Example For Students
The Patriot Movie: Plot Summary Essay The movie The Patriot takes place in South Carolina in 1776. Benjamin Martin, a French, Indian war hero who is haunted by his past, is a patriot who wants no part in a war with Britian. Meanwhile, his two eldest sons, Gabriel and Thomas, cant wait to go out and kill some Redcoats. When South Carolina decides to go to war with Britain, Gabriel immediately signs up to fight without his fathers permission. But soon, Colonel Tavington, British solder infamous for his brutal tactiks, captures Gabriel and sentences him to be hanged. As Gabriel is taken away, Thomas tries to free him, only to be killed by Tavington, in front of Benjamin. Now, seeking revenge for his sons death, Benjamin leaves behind his 5 other children to bring independence to the 13 colonies. After proving himself on the field of battle in the French and Indian War, Benjamin Martin wants nothing more to do with such things, preferring the simple life of a farmer. We will write a custom essay on The Patriot Movie: Plot Summary specifically for you for only $16.38 $13.9/page Order now But when his son Gabriel enlists in the army to defend their new nation, America, against the British, Benjamin reluctantly returns to his old life to protect his son, now serving under his command, and his family from British tyranny Ashamed of his savagery during the French and Indian War, Benjamin Martin decided he would sit out the American Revolution, while his oldest son Gabriel enlisted minutes after South Carolina joined the fight. He changes his mind when his house is burned down and one of his children is killed by the savage Green Dragoon commander Colonel Tavington. Quickly realizing that traditional 18th century warfare tactics wont work, Martin organizes militiamen into a guerilla unit to harass Cornwallis army long enough to allow the French to arrive. Benjamin Martin is a South Carolina planter who is still haunted by his notoriously brutal past as a soldier in the French and Indian War. When the American Revolution comes, he chooses not to fight for the Continental Army because he wants to protect his
Monday, November 25, 2019
Law of Multiple Alleles
Law of Multiple Alleles Multiple alleles is a type of non-Mendelian inheritance pattern that involves more than just the typical two alleles that usually code for a certain characteristic in a species. With multiple alleles, that means there are more than two phenotypes available depending on the dominant or recessive alleles that are available in the trait and the dominance pattern the individual alleles follow when combined together. Gregor Mendel only studied traits in his pea plants that showed simple or complete dominance and had only two alleles that could contribute to any one trait the plant showed. It wasnt until later that it was discovered that some traits can have more than two alleles that code for their phenotypes. This allowed many more phenotypes to be visible for any given trait while still following Mendels Laws of Inheritance. Most of the time, when multiple alleles come into play for a trait, there is a mix of types of dominance patterns that occur. Sometimes, one of the alleles is completely recessive to the others and will be masked by any of those that are dominant to it. Other alleles may be co-dominant together and show their traits equally in the phenotype of the individual. There are also some cases where some alleles exhibit incomplete dominance when put together in the genotype. An individual with this type of inheritance connected to its multiple alleles will show a blended phenotype that mixes both of the alleles traits together. Examples of Multiple Alleles The human ABO blood type is a good example of multiple alleles. Humans can have red blood cells that are of type A (IA), type B (IB), or type O (i). These three different alleles can be combined in different ways following Mendels Laws of Inheritance. The resulting genotypes make either type A, type B, type AB, or type O blood. Type A blood is a combination of either two A alleles (IA IA) or one A allele and one O allele (IAi). Similarly, type B blood is coded for by either two B alleles (IB IB) or one B allele and one O allele (IBi). Type O blood can only be obtained with two recessive O alleles (ii). These are all examples of simple or complete dominance. Type AB blood is an example of co-dominance. The A allele and the B allele are equal in their dominance and will be expressed equally if they are paired together into the genotype IA IB. Neither the A allele or the B allele is dominant over each other, so each type is expressed equally in the phenotype giving the human an AB blood type.
Thursday, November 21, 2019
Helicobacter pylori in gastrointestinal disorders Essay
Helicobacter pylori in gastrointestinal disorders - Essay Example pylori is the production of urease (Graham & Sung, 2006). H.pylori infection is typically acquired in childhood, and affects children by the age of 10-years in developing countries (Graham & Sung, 2006). In developed countries, an age-related increase in prevalence of the disease is seen (Graham & Sung, 2006). Humans are the primary reservoir of infection and the primary mode of transmission is person to person (Graham & Sung, 2006). Oral-to-oral transmission is supported by the finding of H. pylori in dental plaque and saliva by culture and polymerase chain reaction (PCR) while fecal-to-oral transmission is supported by the finding of H. pylori in stool by culture and PCR (Graham & Sung, 2006). Gastro-oral transmission is also possible but more evidence is needed (Graham & Sung, 2006). The ability of H. pylori to colonize and damage gastric mucosal cells is determined by the immune gene polymorphisms of the host and the gastric acid secretion (Kusters, van Vliet, Kuipers, 2006). In addition, bacterial virulence factors like the cytotoxin-associated gene pathogenicity island-encoded protein (CagA) and the vacuolating cytotoxin (VacA) help in the colonization (Kusters, van Vliet, Kuipers, 2006) and also triggers the host inflammatory response by activating the nuclear factor kappaB-dependent gene transcription (Zarrilli, Ricci, Romano, 1999). ââ¬Å"Urease and/or motility of the bacterium, presence of lipopolysaccharide (LPS) and various bacterial enzymesâ⬠are also known to affect the colonization (Lee, 2005.) The healing of the gastric mucosa is also impaired ââ¬Å"through inhibition of epidermal growth factor receptor-dependent signal transduction pathways and induction of apoptosisâ⬠(Zarrilli, Ricci, Romano, 1999). H. pylori infection also may lead to the progression from chronic gastritis to gastric adenocarcinoma (Zarrilli, Ricci, Romano, 1999). The ââ¬Å"cag pathogenicity island (cagPAI), which encodes the type IV secretion
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